Acute pancreatitis case study

De Castro, Richelle Sandriel C.

Acute pancreatitis case study

White blood cells; RBC: Red blood cells; Hb: Fasting blood sugar; T. Blood urea nitrogen; Cr: High density lipoprotein; LDL: Low density lipoprotein; LDH: Anterior-posterior diameter of the visible body portion of the pancreas is in the upper normal range about Slit-like fluid collection arrow in the perisplenic area was consistent with acute pancreatitis; C: A normal gallbladder and no evidence of intra- or extrahepatic duct dilatation were noted.

The patient was managed with intravenous fluids, analgesics and by nil per oral. We monitored the fetus using the non-stress test based on amniotic fluid volume.

The test results for the fetus were satisfactory for gestational age. The mother improved with this supportive treatment within two days and started a soft diet without any symptoms.

The patient was discharged on the 4th hospital day in good health. The patient was followed for 10 d after discharge and was determined to be in good health.

The patient has maintained her health and had not experienced a recurrence of acute pancreatitis when assessed at 10 d after discharge and 15 d postpartum. The precipitating factors included 4 cases of hypertriglyceridemia and 1 case of alcohol-induced pancreatitis; however, there have been no case reports of acute idiopathic pancreatitis during pregnancy in South Korea.


The etiological associations of acute Acute pancreatitis case study during pregnancy are similar to those in the general population. Acute pancreatitis in pregnancy is most often associated with gallstone disease or hypertriglyceridemia[ 1 ].

Several cases of acute idiopathic pancreatitis in pregnancy have been reported worldwide[ 4 - 6 ]. Geng et al[ 7 ] reported that The relationship between acute pancreatitis and pregnancy remains unclear. Pregnancy does not primarily predispose pregnant woman to pancreatitis, but it does increase the risk of cholelithiasis and biliary sludge formation.

Theoretical explanations for the association between pregnancy and biliary tract diseases include an increased bile acid pool size, percentage of cholic acid, cholesterol secretion and decreased enterohepatic circulation. Moreover, progesterone in pregnancy provokes gallbladder volume increase and slow emptying, induces bile stasis in the duodenum and then increases reflux[ 1 ].

Estrogens increase cholesterol secretion and minimally alter gallbladder function. Also, in the third trimester, an enlarged uterus and increased intra-abdominal pressure on the biliary duct bring about acute pancreatitis more frequent[ 8 ]. Hyperlipidemia is the second most common causative factor of acute pancreatitis, and pregnancy increases the level of serum cholesterol, causes biliary stasis and induces gallstone formation.

Hypertriglyceridemia may also directly cause acute pancreatitis. Plasma triglycerides increase to times in pregnancy, principally in the third trimester, due to increased triglyceride-rich lipoprotein production and decreased lipoprotein lipase activity[ 910 ].

In women with abnormal lipoprotein metabolism, this can lead to severe hypertriglyceridemia precipitating pancreatitis. Other etiological factors for acute pancreatitis include alcohol abuse, diabetes mellitus, and, rarely, hyperparathyroidism, connective tissue diseases, abdominal surgery, infections or iatrogenic sources such as diuretics, antibiotics and antihypertensive drugs.

Laboratory tests are essential for diagnosing acute pancreatitis and the tests can include serum amylase, lipase, complete blood count, serum triglycerides, calcium and liver function tests. Abdominal ultrasound is an ideal imaging technique for diagnosing acute pancreatitis in pregnancy because it has no associated radiation risk and is useful for detecting dilated pancreatic ducts and pseudocysts[ 13 ].

However, it is difficult to diagnose acute pancreatitis in pregnancy by ultrasound because an enlarged uterus and combined ileus make a pancreas shadow invisible.

Recently, additional useful imaging tools have been developed to detect acute pancreatitis in pregnancy. Abdominal ultrasound, computed tomography CTendoscopic ultrasound and magnetic resonance cholangiopancreatography MRCP are available for diagnosing a biliary etiology for acute pancreatitis.

The usefulness of CT is substantially restricted in pregnancy because of the potential radiation exposure to the fetus.

Signs and Symptoms of AP

Conservative therapy for acute pancreatitis, which includes gastric decompression, antispasmodic drugs, and antibiotics, is the first choice therapy, and the efficacy of surgical treatment remains controversial.Case Report INTRODUCTION Chronic calcifying/calcific pancreatitis (CCP) is a special form of chronic pancreatitis that tends to calcify or is associated with pancreatic lithiasis.

Assessment & Management of Acute Pancreatitis Huey Cheah Resident Rounds. Oct 21, Case Study: Mr. TR z Mr. TR – 55 y/o male, taxi driver presents to ED with 8 hr h/o severe epigastric pain radiating to back z “acute pancreatitis with diffuse pancreatic ascites. .

Acute pancreatitis case study

pancreatitis to have repeated episodes of acute pancreatitis. Nelms Todays Dietitian Signs & Symptoms Abdominal pain Back pain Nausea Vomiting Fever Swelling of the abdomen Rapid pulse High/low blood pressure Microsoft PowerPoint - Case Study chronic pancreatitis Author. Background DK camps over the weekend and drinks a moderate amount of alcohol.

The following Monday, DK starts to experience abdominal discomfort, nausea, vomiting, fever, and chills. Acute Pancreatitis in a Dog: A Case Report By Robert R.

Acute pancreatitis - Wikipedia

Spencer* Introduction Acute pancreatitis is an important dis­ ease of the canine which is seen periodic­. last study was limited by the lack of both a non ‐ contrast ¾Pros – Non‐invasive and no use of IV contrast – Ability to better characterize fluid collections (acute collection vs.

abscess, necrosis, hemorrhage, pseudocyst) • Acute Pancreatitis is a common illness with.

Doxycycline not to blame for acute pancreatitis - Daniel Cameron, MD, MPH